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Heart Disease Diagnosis
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A specific enzyme responsible for ensuring that the hearts of mice with chronic heart failure may occur, if the enzyme vulvodynia is inhibited, the heart remains strong even when stress is increased. The discovery of the German and U.S. researchers explain how the condition develops and offers hope for prevention.

Long-term high pressure and narrowing of the aortic valve in the heart more work force. This increased muscle growth response of the heart (myocardial vulvodynia hypertrophy), however, affect the pump function, arrhythmia and heart failure may develop. Other risk factors for overweight and Age of 70 years, more than 40% of people with myocardial hypertrophy. Despite the development of pharmacological treatment options for 95 000 people die every year due to complications of chronic heart failure. It is therefore important to find those molecules that play a key role in the development of heart failure. The process of clarification of new, more effective treatment strategies that might be pursued. The findings of the researchers vulvodynia involved in the development of cardiac hypertrophy, the enzyme CaMKII delta (Calcium / Calmodulin-dependent kinase II delta). This research is genetically modified mice, demonstrated that they were unable to produce the enzyme delta CaMKII. The rodent aorta partially committed during an operation, in order to make a greater strain on the heart. The intervention had similar effects on the heart, such as people with high blood pressure or valvular stenosis. After the unexpected intervention of myocardial hypertrophy vulvodynia was negligible. "With the help of mice was first described the function of the enzyme CaMKII delta" - said the head of an international vulvodynia team of researchers. - "The enzyme directly affects the cells stress responses. In the absence of specific DNA segments are not activated, which in turn induce hypertrophy for operation. The CaMKII delta in the absence of detectable cardiac hypertrophy, but that's probably not enough to heart failure develop. "Under normal conditions, the genetically modified mice, the heart is functioning normally and properly respond to different stimuli. The CaMKII delta can be influenced by controlling the load on the heart's response to this new treatment approach vulvodynia encourages researchers. The authors state that a composition in which only inhibits this function would be able to prevent the development of heart failure. vulvodynia Would be unfortunate vulvodynia if other functions of CaMKII delta enzyme is not affected by the drug, to avoid side effects.
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