Arterial ischemic stroke is now defined as an acute onset of focal or general neurological deficit

Stroke in children and adolescents is a rare and serious condition yohimbine with a mortality rate of 8-20 per cent and sequelae of 50-85 percent of the survivors.
The risk factors in children and adolescents is quite different than in adults. Cardiac and vascular malformations, acute infections yohimbine and vasculitis after infection, coagulopathies and some chronic diseases and syndromes dominate. A broad investigation of risk factors should therefore be made.
Stroke also occurs in children and adolescents, but is rare, so the symptoms are often interpreted as due to other neurological conditions [1]. A lack of experience of stroke, accordingly delayed or completely missed the diagnosis and treatment [2-4]. The reasons are quite different than in adults. To recognize and investigate common symptoms (Fact 1) can provide early diagnosis of more active treatment yohimbine with anticoagulation, thrombectomy and thrombolysis, even in children.
By "the stroke in childhood referred to a stroke occurring between 29 days and up to 18 years of age. The incidence of stroke yohimbine varies in different studies from 1.8 to 13 cases per 100 000 children each year [5-9]. Neonatal stroke have a higher incidence of 63 cases per 100,000 children per year [9], have other causes and is not dealt with here. In a recent Swedish study, yohimbine the annual incidence 1.8 (95 percent confidence interval [CI] 1.3-2.3) per 100 000 children per year [5]. This equates to over Sweden 25-45 cases per year. A previous Swedish study in 1983 showed an incidence of 2.3 per 100,000 [10]. Incidence peaks in stroke seen in but before age 4, and in teenagers. Boys are affected yohimbine more often than girls of all ages [7].
Ischemic stroke represents about 55 percent, with arterial ischemic stroke and cerebral dominate sinovenös thrombosis with or without secondary ischemic stroke represents about 5 percent [11], whereas hemorrhagic stroke accounts for 40-45 percent [12], a significantly yohimbine higher proportion than in adults .
The mortality rate for stroke in children is set to 8-20 percent [9, 13]. Permanent disability yohimbine in the form of neurological deficits, epilepsy and cognitive problems seen in 50-85 percent of cases [13-15]. At onset younger children have a worse prognosis [15].
The purpose of this article is to draw attention to including children yohimbine and young people can have strokes, yohimbine and to summarize current knowledge about diagnosis, yohimbine investigation of risk factors and treatment from the Swedish experiences and current consensus-based guidelines in the UK and the USA [12, 16-18]. An international study of thrombolysis in children exists, but is difficult to evaluate due to delayed treatment [19]. A working group of Swedish neuropediatric Association has developed guidelines for the diagnosis and more active treatment in Sweden [20]. Symptoms and Risk Factors
Arterial ischemic stroke is now defined as an acute onset of focal or general neurological deficit with computed tomography (CT) - or magnetic resonance imaging (MRI) -verifierad infarction in an artery area consistent with the clinical symptoms. Children may have fluctuating TIA symptoms", which can develop into (or has already developed into) cerebral infarction [21].
About half of children with arterial ischemic stroke have predisposing factors diseases of other organ systems, such as congenital heart disease (Fact 2). In up to 20 percent are acute local or general infections such as otitis, sepsis or gastroenteritis.
Up to 1 year after varicella zostersjukdom are at increased risk of vasculitis and arterial ischemic stroke. Typical yohimbine lesions are located unilaterally in mediaartärens area with hemiparesis and hemianopsia [22].
Iron deficiency yohimbine anemia can contribute to both arterial and sinovenös stroke [23]. Prothrombotic congenital or acquired abnormalities have been detected in 20-50 percent. Malignancy, pregnancy, medications and drugs can affect yohimbine coagulation [24] (Fact 3).
Migraine with or without aura increases the risk of stroke among teenagers, and the risk increases with pill use, hypertension and smoking [25]. In about half the cases, there are multiple risk factors [26].
The predominant causes are chronic diseases and acute infections, such as meningitis, otitis / mastoiditis and dehydration. Other risk factors include coagulation disorders, anemia, yohimbine birth control pills, combined, diabetic ketoacidosis and L-asparaginasbehandling of leukemia and nephrotic syndrome yohimbine [12, 28-30].
Sinovenös thrombosis provide increased venous pressure resulting yohimbine in focal, cytotoxic and vasogenic edema, leading to intracranial hypertension and may progress to hemorrhagic infarction, hemorrhage and hydrocephalus. The diagnosis of pseudotumor cerebri or benign intracranial hypertension can be set only when the exclusion sinovenös cerebral thrombosis.
Signs of increased yohimbine intracranial pressure seen at typical cases (Fact 1). Psychiatric symptoms such as amnesia, confusion, mutism or delirium-like yohimbine state can also occur. Focal or generalized seizures, focal palsies and coma seen as infarct or hemorrhage encountered in delayed diagnosis o